medical SLUG: person-no-longer-breathing-for-some-reason
Threat Level 5 — Leave Faster

Person No Longer Breathing for Some Reason

Sudden cardiac arrest recognition, high-performance CPR, and automated external defibrillator (AED) deployment

// IMMEDIATE ACTIONS

Execute these steps in order:

  1. 01
    CHECK RESPONSIVENESS AND BREATHING (10 SEC MAX)

    Quickly assess for responsiveness and normal breathing:

    • ├─ Shake the shoulders firmly and shout: "Are you okay?"
    • ├─ Tilt the head back gently and lift the chin to open airway; look and listen for normal breathing (max 10s).
    • └─ Agonal gasps = Cardiac Arrest: Slow, snoring, irregular gasps are NOT normal breathing; act immediately.
  2. 02
    CALL 911 / 112 AND GET AN AED

    Put phone on speakerphone with emergency dispatch and direct a bystander to bring an Automated External Defibrillator.

  3. 03
    START CHEST COMPRESSIONS IMMEDIATELY

    Deliver high-quality chest compressions in the center of the chest:

    • ├─ Heel of one hand on lower breastbone, second hand interlocked on top, elbows locked straight.
    • ├─ Push hard and fast: 5 to 6 cm depth at 100 to 120 compressions per minute (tempo of "Stayin’ Alive").
    • ├─ Allow complete chest recoil between compressions without leaning.
    • └─ Compression-Only CPR is fully effective; if trained, alternate 30 compressions : 2 rescue breaths.
  4. 04
    ATTACH AED AS SOON AS IT ARRIVES

    Power on the AED and follow clear voice prompts without delay:

    • ├─ Apply adhesive pads to bare chest: Upper right collarbone and lower left side below armpit.
    • ├─ Stand clear: Ensure NO ONE touches the person during analysis or shock delivery.
    • └─ Resume chest compressions immediately after shock or if no shock is advised.

// CRITICAL MISTAKES (DO NOT)

  • ✕ Do not waste time checking for a pulse
    WHY: clinical studies prove laypersons cannot reliably detect carotid pulses in emergencies; absence of response and normal breathing is the sole criteria
  • ✕ Do not hesitate to start CPR if the person is taking occasional gasping breaths
    WHY: agonal respiration occurs in ~50% of cardiac arrests and is a direct sign of cerebral hypoxia
  • ✕ Do not remove the AED pads after a shock is delivered
    WHY: the device must continuously monitor the heart rhythm every 2 minutes
  • ✕ Do not interrupt chest compressions for more than 10 seconds for any reason.
  • ✕ Do not fear hurting the patient or breaking ribs
    WHY: rib fractures heal; untreated cardiac arrest is 100% fatal within 8 minutes
MEMORY HOOK (THE ONE THING TO REMEMBER)
“Call 911/112, push hard and fast in the center of the chest (100–120 bpm, 5–6 cm deep), and use an AED immediately.”

The human circulatory system is an entirely closed hydraulic circuit. The left ventricle pumps pressurized oxygenated blood into the aorta, maintaining a cerebral perfusion pressure of at least 60 mmHg. When the heart abruptly ceases mechanical pumping—typically triggered by chaotic ventricular fibrillation (VF) or pulseless electrical activity (PEA)—systemic arterial blood pressure crashes to zero in less than three seconds.

Without active arterial perfusion, consciousness is lost within 10 to 15 seconds. Irreversible ischemic neuronal death in the cerebral cortex begins in under four minutes.

Recognizing Cardiac Arrest (The Agonal Breathing Trap)

The single greatest point of failure in lay resuscitation is misinterpreting agonal respiration. In approximately 50% of sudden cardiac arrests, the hypoxic brainstem continues firing sporadic, reflexive electrical impulses to the diaphragm for several minutes.

This presents as heavy, irregular, snoring, or gasping sounds occurring every 10 to 15 seconds without chest movement. This is not breathing; it is the death rattle of a dying brainstem. If a person is unresponsive and not breathing normally and rhythmically, assume cardiac arrest and begin compressions immediately.

The Mechanics of High-Quality Chest Compressions

Manual chest compressions function as an external mechanical pump, squeezing the heart between the sternum and thoracic spine to generate artificial forward cardiac output (typically 20% to 30% of normal baseline perfusion):

  1. Depth (5 to 6 cm): Compressions shallower than 5 cm fail to generate sufficient left ventricular ejection fraction to perfuse coronary and carotid arteries. Compressions deeper than 6 cm increase thoracic trauma without increasing cardiac output.
  2. Rate (100 to 120 beats per minute): Pushing slower than 100 bpm fails to build up cumulative mean arterial pressure. Pushing faster than 120 bpm deprives the heart chambers of diastolic filling time between strokes.
  3. Full Recoil: Leaning on the chest between compressions prevents negative intrathoracic pressure from drawing venous blood back into the right atrium, collapsing artificial perfusion.

Defibrillation (The AED)

Chest compressions generate blood flow, but they almost never terminate ventricular fibrillation on their own. The definitive cure for ventricular fibrillation is an electrical shock delivered by an Automated External Defibrillator (AED). The shock depolarizes the entire myocardial mass simultaneously, resetting chaotic electrical activity and allowing the heart's natural pacemaker (the Sinoatrial Node) to resume coordinated contractions. Every minute of delay before defibrillation reduces survival odds by 7% to 10%.

EMERGENCY DISPATCH NUMBERS

112 reaches fire and emergency medical dispatch directly. 110 is police.

Spotted an error or outdated standard?
// RELATED FIELD NOTES

Additional safety instructions and adjacent hazard knowledge.